Volume 1/ Number 2/ September 2001

 

 

 

 

 

 

 

 

 

 

 

 

 

 

 

 

 

 

 

 

 

 

 

 

 

 

 

 

 

 

 

 

 

 

 

 

 

 

 

 

 

 

 

 

 

 

 

 

 

 

 

 

 

 

 

 

 

 

 

 

 

 

 

 

 

 

FEMORAL ARTERY THROMBOSIS FOLLOWING HEMIARTROPLASTY OF THE HIP:
A Case Report

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Introduction
Case Report
Discussion
References



Abstract: 
Thrombosis of the femoral artery during total hip replacement is a serious limb-threatening complication. Although Iatrogenic damage of the femoral or iliac vessels is reported frequently in total hip arthroplasty, is very rare in partial hip replacement. We report here a case of cementless hemiarthroplasty of the hip complicated with extensive femoral artery thrombosis and subsequent amputation of the limb. The early diagnosis and management of such serious complications is highlighted. 

Introduction: 

Thrombosis of the femoral and common iliac vessels following hemiarthroplasty of the hip joint is a very rare complication. Most of the cases of vascular complications in hip arthroplasty are reported in total hip replacement and the literature has scarce reports of vascular complications in hemiarthroplasty of the hip. 
We here report a case of extensive femoral artery thrombosis following the Austin Moore hemiarthroplasty of the hip in a 75 years old man.

Case Report:

A 75 years old man presented in the Emergency section of our hospital with a history of fall on the left hip. On examination the patient's general condition was good with no systemic disorder. The left lower limb was externally rotated and abducted. There was no neuro-vascular deficit, peripheral pulses were palpable normally on both sides. X-ray of the left hip confirmed the diagnosis of Subcapital Fracture of the Neck of the Femur (Garden's Grade IV). A pre-operative medical check up was done and it was normal, since the patient was an elderly gentleman, prophylactic heparin was administered, Hydralazine was used as vasodilator, this was supplemented along with an antiroloux formation agent PentoxYfyline. The baseline PT, PIT and total blood counts were all with in normal limits. 

The left hip was opened through a modified Hardinge's approach and the head of the femur replaced by an Austin Moore's prosthesis (size 51). The patient did well in the first 24 hours of the surgery and there was no obvious neurovascular deficit. 

On the second post-operative day it was noticed that the patient was not able to flex the knee and there were no ankle and toe movements. On examination, the limb was anesthetic below the knee and dorsalis pedis, posterior tibial and popliteal arteries were not palpable. A check X-ray of the hip showed that the prosthesis was in satisfactory position. The patient was put on peripheral vasodilators and heparin injections. A color Doppler ultrasound of the involved limb was immediately done. It showed (Fig. 1) a thrombus in the left femoral artery with no distinct blood flow down to the dorsalis pedis artery.

Fig.1 Colour Doppler of the left lower limb showing thrombosis of the femoral artery. Note the absence of blood flow in distal vessels.

ECG, Doppler, Echo and all other investigations to rule out other causes of emboli from other regions of the cardiovascular system were done and found to be non-contributory. An emergency exploration of the femoral artery was done and a thrombus about 20 cm long was removed using a Fogarty Catheter. Fasciotomy of the left leg compartments was also done. There was no objective improvement in the neurovascular status of the limb in the post-operative period. Subsequently the limb became gangrenous and ultimately an above knee amputation had to be done. 

Discussion:

Extensive femoral and iliac artery thrombosis, during total hip replacement has been reported frequently in world literature. According to Fruhwrith et al(l), the vascular complications in total hip replacement usually occur while relocating the head of a cemented prosthesis. They have classified the vascular injuries during total hip arthroplasty into lacerations, thrombosis, pseudo- aneurysms and arterio-venous fistula. Bindewald et al (2) feel that injuries of the femoral vessels seem to be preventable if the Hohmann-retractor is carefully placed on the anterior margin of the acetabular rim under digital control. Furthermore drilling damage of the iliac vessel appears to be avoidable. We had not used the Hohmann retractor in our case. 

The activation of clotting cascade leading to deep vein thrombosis in total hip arthroplasty has been assigned as an important cause of vascular complication in total hip arthroplasty (3).

The risk of vascular complications is definitely increased in patients undergoing revision hip arthroplasty. Revision total hip arthroplasty is now considered as a definite risk factor for iatrogenic vascular injury (4). 

The early diagnosis and management of this limb threatening complication is a must and should be realised by every surgeon. In our case there was a delay of around 24 hours before the diagnosis of femoral artery thrombosis could be established. As a result of this delay, the limb could not be saved despite adequate surgery. According to Fruhwrith et al (1) the diagnosis should usually be made on the operating table itself. They report a case of iatrogenic femoral thrombosis presenting after three weeks with a femoral pseudo-aneurysm. 

We were not able to find any case of vascular complication in partial hip replacement in world literature. Our case report highlights the importance of being aware of the occurrence of this dreaded complication even in hemiarthroplasty of the hip, which is a far more common surgery performed in this part of the world. 

References: 

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