Pages (3): [< 1 2 3 > ]

1. Previous DVT/PE 10 Years back

2. Travel by economy class, buses or cars for more than four hours.

3. History of recurrent abortions and miscarriages

4. Family History of idiopathic DVT/PE

5. Arthroscopic knee surgery one-month back

1. There is no clinical finding pathognomonic for PE, nor is there a clinical picture that can rule it out

2. Dyspneoa and Tachypneoa are the most common presentation in angiographically proven PE

3. Pleuritic chest pain is the commonest clinical presentation in P.E

4. The classical triad of (Dyspneoa, Haemoptysis and Pleuritic chest pain) occurs in 70% of angiographically proven PE

5. P.E could be detected in 50% of radiologically proven proximal DVT.

1. Indeterminate V /Q scans are common in patients with COPD and bronchial asthma.

2. Normal V/Q scans cannot rule out PE in 4% of cases.

3. Matching V /Q scan lung lesions indicates further invasive investigations if the pre-test likelihood of PE is high.

4. Two or more mis-matched lung segmental defects make the V /Q scan of high probability for PE.

5. V/Q scan cannot be performed on a hemodynamically unstable patients or patients on a ventilator

1. High isolated PTT is common in patients with Lupus Anticoagulant

2. D-Dimer test is both highly sensitive and specific for VTE

3. WBC >20,000 rules out PE

4. CBC, PT, PTT should be monitored at least daily in any patient on IV Heparin

5. Specific lab. Tests for diagnosis of HIT is not widely available and takes time

1. It doesn't make a significant difference if coumadin (Warfarin) was started with Heparin on the first or third day.

2. It is recommended to continue both IV heparin and Coumadin for at least 24-48 hours after INR reaches a therapeutic level.

3. Unfractionated IV Heparin is superior to LMWH for treating VTE

4. INR of 4.0-5.0 is a safe therapeutic range of Warfarin

5. Patients with previous history of exposure to unfractionated Heparin have higher chance to develop HIT

1. New onsetA. Fib. is pathgnomonic for PE

2. New onset RBBB is pathgnomonic. for PE

3. S1Q3T3 is found in 90% of angiographically proven PE

4. Normal ECG cannot rule out PE

5. No single ECG changes is specific for PE

1. This Patient should have had ascending venogram and lor pulmonary angiogram before starting her on I. V Heparin

2. This Patient doesn't need life-long treatment with anticoagulants

3. This young lady cannot get pregnant anymore.

4. Ancrod (IV freshly prepared Snake Venom) could have been used to treat her illT

5. If she ever gets pregnant she should stop coumadin on discovering pregnancy and be started on LMWH .

1. HIT (Heparin -Induced Thrombocytopenia) was highly suspicious

2. Haematuria was secodery to renal infarction secondary to renal artery Nein thrombosis

3. Platelets should have been transfused to correct her thrombocytopenia

4. IVC filter was not indicated

5. Surgical Thrombectomy and interruption of IVC by James De Weiss clip or plication could be another alternative for IVC filter

1. High (proximal) DVT is defined as clots involving the femoral vein proximal to the popliteal vein

2. Venous Doppler ultrasound is now the gold standard test for DVT

3. Pulmonary angiogram is absolutely out as it is an invasive, expensive and not readily available test

4.Performing ascending venogram on the symptomatic leg only, is faster and safer than bilateral ascending venograms

5.Helical (spiral) CAT scan is highly sensitive for massive and sub massive PE

1.Patients with idiopathic (No Risk Factors) VTE should be screened for thrombophilias

2. Families of patients with protein S or protein C deficiency should be screened

3. Significant percentage of patients with history of recurrent abortions or miscarriages could have positive L.A factor

4. Protein C, Protein S, are Vit. K dependant proteins

5. Blood for thrombophilias screening should be collected before starting Coumadin

Pages (3): [< 1 2 3 > ]